NAVIGEN, INC. — Department of Health and Human Services SBIR Phase I: NHLBI

NAVIGEN, INC. — SBIR Phase I award from Department of Health and Human Services.

Phase I SBIR feasibility signal

  • Phase I awards fund proof-of-concept work. For capture teams, they mark early interest from Department of Health and Human Services in a technical approach.
  • Watch for Phase II follow-ons from the same firm/topic family — that conversion path is where budgets and transition pressure rise.
  • Obligated amount $268,075. Cross-check similar awards in the same agency and technology tags for going-rate context.
  • Topic code NHLBI links this award to a solicitation family — search the same topic stem for incumbents and recompete timing.

Informational capture context from public federal data — not legal or bid advice.

Amount
$268,075
Agency
Department of Health and Human Services · National Institutes of Health
Program / Phase
SBIR · Phase I
Topic
NHLBI
Solicitation
PA18-574
NAICS
Place of performance
UT
Period
2019-09-24 → 2020-09-19

Description

SUMMARYInfluenzavirusinfectionisoneofthemostcommonacuterespiratorydiseasesandremainsoneofthelargestdiseaseburdensonhumanswithsevereprimaryviralpneumoniaoftenresultingindeathThepathogenicityofinfluenzaincludesahostresponsethatcanincludeaseveresystemicinflammatoryreactioncytokinestormandensuingvascularleakVascularleakisafundamentalelementinthepathogenesisofcirculatoryshockandmultipleorganfailurewhichcanleadtodeathininfluenzapatientsThelaboratoriesofDrDean Licofounder and former Chief Scientific Officer of Navigenand DrShannon Odelbergalong withNavigenhavedemonstratedthatvascularleakassociatedwithinflammationismediatedbyactivationofthesmall GTPaseARFa common nodal point in the signaling pathways of several inflammatory cytokines andgrowth factorsNavigen s objective is to reduce morbidity and mortality associated with influenza infections bymodifyingthishostresponseSpecificallyweproposetoevaluatetheefficacyofoursmallmoleculeARFinhibitorswiththehypothesisthatinhibitionofARFwillreducevascularleakelicitedbytheinfectionwhilehaving no adverse effect on immunitybased clearance of the virusNARRATIVEInfluenzainfectionremainsoneofthelargestdiseaseburdensonhumanswithsevereprimaryviralpneumoniaoftenresultingindeathThepathogenicityincludesahostresponsethatcanincludeaseveresystemicinflammatoryreactionandensuingvascularleakNavigenhasdemonstratedthatvascularleakassociated with inflammation is mediated by activation of the small GTPase ARFand proposes to evaluate itssmallmolecule ARFinhibitors in a mouse model of H Ninfection